A study reported in the journal Apoptosis just weeks ago indicates that this controversial vaccine normally injected into newborns within hours of birth induces liver damage primarily due to the presence of the toxic vaccine adjuvant aluminum hydroxide.
Vaccine adjuvants like aluminum hydroxide are the dirty little secret that the scientific community doesn’t want the public to know about. Their purpose is to stimulate and heighten the immune response to the vaccine itself.
In this new study, aluminum hydroxide induced loss of mitochondrial integrity and cell death in mouse liver cells exposed to low doses of the Hepatitis B vaccine containing the toxic adjuvant.
The mitochondria is the “powerhouse” of a cell where energy is created and cellular respiration occurs. It is not surprising that compromising the mitochondria of liver cells results in outright death of the cell itself.
It’s not just the young developing livers of children that suffer from the toxic Hep B. The JournalHepatogastroenterology reported in 2002 that the Hepatitis B vaccine was statistically associated with increased risk for hepatitis, gastrointestinal disease, and liver function test abnormalities in adult females.
Did you get that?
The Hepatitis B vaccine is statistically associated with increased risk for hepatitis in adults!
A more recent study in June 2011 reported by the journal Molecular Biology Reports indicates that damage from Hep B is likely caused by alteration in the expression of 144 genes in mouse livers within one day of vaccination, 7 of which are associated with inflammation and metabolism.
The Only Sane Answer is Rejection of the Hepatitis B Vaccine
The thing that makes absolutely zero sense is why the controversial and very questionable Hepatitis B vaccine is still being recommended as standard for all newborns within hours of birth especially given that a newborn’s liver does not even begin functioning until several days later.
With no functioning liver to assist with detoxification, the damaging effects of the toxic Hep B ingredients would no doubt be magnified with the risk of irreparable harm to the newborn great.
If you are a parent with a baby on the way, please do your research and learn the extreme danger of the Hepatitis B vaccine to your newborn.
There is simply no logical reason to vaccinate all newborns against a disease that is primarily sexually transmitted or contracted via the used needles of drug addicts. Better to screen mothers preemptively rather than vaccinate all newborns with this dangerous drug cocktail that threatens their long term health.
Key Lab of Agricultural Animal Genetics, Breeding, and Reproduction of Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan, People's Republic of China. Heyam68_hamza@yahoo.com
Vaccines can have adverse side-effects, and these are predominantly associated with the inclusion of chemical additives such as aluminum hydroxide adjuvant. The objective of this study was to establish an in vitro model system amenable to mechanistic investigations of cytotoxicity induced by hepatitis B vaccine, and to investigate the mechanisms of vaccine-induced cell death. The mouse liver hepatoma cell line Hepa1-6 was treated with two doses of adjuvanted (aluminium hydroxide) hepatitis B vaccine (0.5 and 1 μg protein per ml) and cell integrity was measured after 24, 48 and 72 h. Hepatitis B vaccine exposure increased cell apoptosis as detected by flow cytometry and TUNEL assay. Vaccine exposure was accompanied by significant increases in the levels of activated caspase 3, a key effector caspase in the apoptosis cascade. Early transcriptional events were detected by qRT-PCR. We report that hepatitis B vaccine exposure resulted in significant upregulation of the key genes encoding caspase 7, caspase 9, Inhibitor caspase-activated DNase (ICAD), Rho-associated coiled-coil containing protein kinase 1 (ROCK-1), and Apoptotic protease activating factor 1 (Apaf-1). Upregulation of cleaved caspase 3,7 were detected by western blot in addition to Apaf-1 and caspase 9 expressions argues that cell death takes place via the intrinsic apoptotic pathway in which release of cytochrome c from the mitochondria triggers the assembly of a caspase activation complex. We conclude that exposure of Hepa1-6 cells to a low dose of adjuvanted hepatitis B vaccine leads to loss of mitochondrial integrity, apoptosis induction, and cell death, apoptosis effect was observed also in C2C12 mouse myoblast cell line after treated with low dose of vaccine (0.3, 0.1, 0.05 μg/ml). In addition In vivo apoptotic effect of hepatitis B vaccine was observed in mouse liver.